Akhter, S. A., Luttrell, L. M., Rockman, H. A., Iaccarino, G., Lefkowitz, R. J., & Koch, W. J. (1998). Targeting the receptor-Gq interface to inhibit in vivo pressure overload myocardial hypertrophy.Science, 280(5363), 577-574.
Alessi, D. R., James, S. R., Downes, C. P., Holmes, A. B., Gaffney, P. R., Reese, C. B., & Cohen, P. (1997). Characterization of a 3-phosphoinositide-dependent protein kinase which phosphorylates and activates protein kinase Bα. Current Biology, 7(4), 261-269.
Bradley, T. J., Slorach, C., Mahmud, F. H., Dunger, D. B., Deanfield, J., Deda, L., … & Moineddin, R. (2016). Early changes in cardiovascular structure and function in adolescents with type 1 diabetes. Cardiovascular Diabetology, 15(1), 31.
Buss, S. J., Riffel, J. H., Malekar, P., Hagenmueller, M., Asel, C., Zhang, M., … & Hardt, S. E. (2011). Chronic Akt blockade aggravates pathological hypertrophy and inhibits physiological hypertrophy. American Journal of Physiology-Heart and Circulatory Physiology, 302(2), H420-H430.
Chis, I. C., Baltaru, D., Maier, M., Muresan, A., & Clichici, S. (2013). Effects of quercetin and chronic (training) exercise on oxidative stress status in animals with streptozotocin-induced diabetes. Bulletin of University of Agricultural Sciences and Veterinary Medicine Cluj-Napoca. Veterinary Medicine, 70(1), 31-39.
DeBosch, B., Treskov, I., Lupu, T. S., Weinheimer, C., Kovacs, A., Courtois, M., & Muslin, A. J. (2006). Akt1 is required for physiological cardiac growth. Circulation, 113(17), 2097-2104.
De Luca, A., Stefani, L., Pedrizzetti, G., Pedri, S., & Galanti, G. (2011). The effect of exercise training on left ventricular function in young elite athletes. Cardiovascular Ultrasound, 9(1), 27.
De Nigris, V., Pujadas, G., La Sala, L., Testa, R., Genovese, S., & Ceriello, A. (2015). Short-term high glucose exposure impairs insulin signaling in endothelial cells. Cardiovascular Diabetology, 14(1), 1-7.
Deshmukh, A., Coffey, V. G., Zhong, Z., Chibalin, A. V., Hawley, J. A., & Zierath, J. R. (2006). Exercise-induced phosphorylation of the novel Akt substrates AS160 and filamin A in human skeletal muscle. Diabetes, 55(6), 1776-1782.
Dillmann, W. (2010). Cardiac hypertrophy and thyroid hormone signaling. Heart Failure Reviews, 15(2), 125-132.
Dorn, G. W., & Force, T. (2005). Protein kinase cascades in the regulation of cardiac hypertrophy. The Journal of Clinical Investigation, 115(3), 527-537.
Engelman, J. A., Luo, J., & Cantley, L. C. (2006). The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism. Nature Reviews Genetics, 7(8), 606-619.
Falcao-Pires, I., Hamdani, N., Borbély, A., Gavina, C., Schalkwijk, C. G., Van Der Velden, J., … & Leite-Moreira, A. F. (2011). Diabetes mellitus worsens diastolic left ventricular dysfunction in aortic stenosis through altered myocardial structure and cardiomyocyte stiffness. Circulation, 124(10), 1151-1159.
Fruman, D. A., & Rommel, C. (2014). PI3K and cancer: lessons, challenges and opportunities. Nature Reviews Drug Discovery, 13(2), 140-156.
Henriksen, E. J. (2002). Invited review: Effects of acute exercise and exercise training on insulin resistance. Journal of Applied Physiology, 93(2), 788-796.
Holloway, T. M., Bloemberg, D., Da Silva, M. L., Simpson, J. A., Quadrilatero, J., & Spriet, L. L. (2015). High intensity interval and endurance training have opposing effects on markers of heart failure and cardiac remodeling in hypertensive rats. PloS One, 10(3), e0121138.
Howlett, K. F., Sakamoto, K., Yu, H., Goodyear, L. J., & Hargreaves, M. (2006). Insulin-stimulated insulin receptor substrate-2–associated phosphatidylinositol 3–kinase activity is enhanced in human skeletal muscle after exercise. Metabolism, 55(8), 1046-1052.
James, S. R., Peter Downes, C., Gigg, R., Grove, S. J., Holmes, A. B., & Alessi, D. R. (1996). Specific binding of the Akt-1 protein kinase to phosphatidylinositol 3, 4, 5-trisphosphate without subsequent activation. Biochemical Journal, 315(3), 709-713.
Kee, H. J., & Kook, H. (2010). Roles and targets of class I and IIa histone deacetylases in cardiac hypertrophy. BioMed Research International, 2011, 1-10.
Kessler, A., Uphues, I., Ouwens, D. M., Till, M., & Eckel, J. r. (2001). Diversification of cardiac insulin signaling involves the p85α/β subunits of phosphatidylinositol 3-kinase. American Journal of Physiology-Endocrinology and Metabolism, 280(1), E65-E74.
Leng, Y., Wang, Z., Tsai, L. K., Leeds, P., Fessler, E. B., Wang, J., & Chuang, D. M. (2015). FGF-21, a novel metabolic regulator, has a robust neuroprotective role and is markedly elevated in neurons by mood stabilizers. Molecular Psychiatry, 20(2), 215-223.
Lew, J. K. S., Pearson, J. T., Schwenke, D. O., & Katare, R. (2017). Exercise mediated protection of diabetic heart through modulation of microRNA mediated molecular pathways. Cardiovascular Diabetology, 16(1), 10.
Li, J., & Brooks, G. (1997). Downregulation of cyclin-dependent kinase inhibitors p21 and p27 in pressure-overload hypertrophy. American Journal of Physiology-Heart and Circulatory Physiology, 273(3), H1358-H1367.
Luo, J., McMullen, J. R., Sobkiw, C. L., Zhang, L., Dorfman, A. L., Sherwood, M. C., ... & Cantley, L. C. (2005). Class IA phosphoinositide 3-kinase regulates heart size and physiological cardiac hypertrophy. Molecular and Cellular Biology, 25(21), 9491-9502.
Ma, Z., Qi, J., Meng, S., Wen, B., & Zhang, J. (2013). Swimming exercise training-induced left ventricular hypertrophy involves microRNAs and synergistic regulation of the PI3K/AKT/mTOR signaling pathway. European Journal of Applied Physiology, 113(10), 2473-2486.
McMullen, J. R., Shioi, T., Huang, W. Y., Zhang, L., Tarnavski, O., Bisping, E., ... & Riggi, L. (2004). The insulin-like growth factor 1 receptor induces physiological heart growth via the phosphoinositide 3-kinase (p110α) pathway. Journal of Biological Chemistry, 279(6), 4782-4793.
Mohammadi, R., Homaee, H., Azarbayjani, M. A., & Baesi, K. (2013). The effect of 12-week resistance training on cardiac hypertrophy, glucose level, insulin, and insulin resistance index in STZ-induced diabetic rats. Qom University of Medical Science, 11(2), 38-45. [Persian]
Omidi, M., & Moghadasi, M. (2018). Effect of 8 weeks aerobic training on pancreatic β-cells function and insulin resistance of female patients with type 2 diabetes. Iranian Journal of Diabetes and Metabolism, 17(2), 79-86.
Pour Haydari, A., & Rahmani Nia, F. (2018). Effects of aerobic training and detraining on body composition, lipid profile and insulin resistance in overweight policemen. Journal of Practical Studies of Biosciences in Sport, 6(11), 85-93. [Persain]
Ramos, J. S., Dalleck, L. C., Tjonna, A. E., Beetham, K. S., & Coombes, J. S. (2015). The impact of high-intensity interval training versus moderate-intensity continuous training on vascular function: a systematic review and meta-analysis. Sports Medicine, 45(5), 679-692.
Rohilla, A., Kumar, P., Rohilla, S., & Kushnoor, A. (2012). Cardiac hypertrophy: a review on pathogenesis and treatment. International Journal of Pharmaceutical Science and Drug Research, 4, 164-167.
Röhling, M., Herder, C., Stemper, T., & Müssig, K. (2016). Influence of acute and chronic exercise on glucose uptake. Journal of Diabetes Research, 2016, 1-33.
Schoenfeld, B. J. (2010). The mechanisms of muscle hypertrophy and their application to resistance training. The Journal of Strength & Conditioning Research, 24(10), 2857-2872.
Shah, A. M., Uno, H., Køber, L., Velazquez, E. J., Maggioni, A. P., MacDonald, M. R., ... & Solomon, S. D. (2010). The inter‐relationship of diabetes and left ventricular systolic function on outcome after high‐risk myocardial infarction. European Journal of Heart Failure, 12(11), 1229-1237.
Sheikhzadeh, F., Khajehnasiri, N., Banan Khojasteh, S. M., GhadiriSoufi, F., Dastranj, A., & Taati, M. (2013). The effect of regular moderate exercise, on cardiac hypertrophy and blood glucose level in diabetic adult male rats. International Research Journal of Applied and Basic Sciencesin Sport, 6(4), 499-503
Shiojima, I., & Walsh, K. (2006). Regulation of cardiac growth and coronary angiogenesis by the Akt/PKB signaling pathway. Genes & Development, 20(24), 3347-3365.
van Heerebeek, L., Hamdani, N., Handoko, M. L., Falcao-Pires, I., Musters, R. J., Borbély, A., ... & Diamant, M. (2008). Response to letter regarding article,Diastolic stiffness of the failing diabetic heart: importance of fibrosis, advanced glycation end products, and myocyte resting tension. Circulation, 117(23), e484-e484.
Waardenberg, A. J., Bernardo, B. C., Ng, D. C., Shepherd, P. R., Cemerlang, N., Sbroggio, M., ... & McMullen, J. R. (2011). Phosphoinositide 3-kinase (PI3K) (p110α) directly regulates key components of the Z-disc and cardiac structure. Journal of Biological Chemistry, 286(35), 30837-30846.
Wallace, T. M., Levy, J. C., & Matthews, D. R. (2004). Use and abuse of HOMA modeling. Diabetes Care, 27(6), 1487-1495.
Walsh, K. (2006). Akt signaling and growth of the heart. Circulation, 113(17), 2032-2034.
Wild, S., Roglic, G., Green, A., Sicree, R., & King, H. (2004). Global prevalence of diabetes: estimates for the year 2000 and projections for 2030. Diabetes Care, 27(5), 1047-1053.
Yu, W., Chen, C., Fu, Y., Wang, X., & Wang, W. (2010). Insulin signaling: a possible pathogenesis of cardiac hypertrophy. Cardiovascular Therapeutics, 28(2), 101-105.
Zare Karizak, S., Kashef, M., Gaeini, A., Nejatian, M. (2017). The comparison of two protocol of interval and continues aerobic training on level of concentric pathologic hypertrophy and cardiac function in patients after coronary artery bypass grafting surgery. Journal of Practical Studies of Biosciences in Sport, 5(9), 9-20. [Persain]
)